Author: Terry Micheal TurnerPublisher:
ISBN:
Category : Cotton
Languages : en
Pages : 50
Book Description
A Genetic Analysis of Gl2, Gl3, and a Gene, Vt., Determining Tolerance to Verticillium Wilt in Cotton
Author: Terry Micheal TurnerPublisher:
ISBN:
Category : Cotton
Languages : en
Pages : 50
Book Description
Genetics of Verticillium Wilt Tolerance in Cotton
Author: Michael Edward DeveyPublisher:
ISBN:
Category : Cotton
Languages : en
Pages : 240
Book Description
ARS-S.
Author: A Quantitative Genetic Study of Verticillium Wilt Resistance Among Selected Lines of Upland Cotton
Author: Kwee-Chong PanMapping of Verticillium Wilt Resistance Genes in Cotton
Author: Yuksel BolekVerticillium Wilt of Cotton ; Proceedings
Author: Work conference on Verticillium wilt of cotton, National cotton pathology research laboratory, College Station, Texas, 1971Publisher:
ISBN:
Category :
Languages : en
Pages : 134
Book Description
Evaluation and Confirmation of a Multi-parent Advanced Generation Inter-cross (MAGIC) Introgressed Line Population for Verticillium Wilt Resistance in Upland Cotton
Author: Gasper MartinezPublisher:
ISBN:
Category :
Languages : en
Pages : 198
Book Description
Upland cotton (Gossypium hirsutum L.) is the most important fiber crop, and the US is the third largest cotton producer after India and China and the largest cotton exporter in the world. Verticillium wilt (VW, caused by Verticillium dahliae Kleb.) is a destructive fungal soil-borne disease in Upland cotton production. High levels of VW resistance exist in Pima cotton (G. barbadense L.) and can be transferred into Upland cotton through interspecific introgression breeding. In a greenhouse study, VW resistance was evaluated in a multi-parent advanced generation inter-cross (MAGIC) introgressed line (IL) population of 712 lines, generated from a Random mated Barbadense Upland Population (RMBUP-C4) derived from five cycles of random mating of crosses between three Upland cotton cultivars and 18 chromosome substitution lines (CSLs) each carrying a pair of Pima cotton chromosome or arm. The objectives of this study were to evaluate VW resistance of the MAGIC IL population in the greenhouse to identify ILs with VW resistance based on a total of four replicated greenhouse tests. In 2016-2017, a total of 712 MAGIC ILs derived from RMBUP-C4 were evaluated for VW resistance in two replicated tests (Test 1 and 2) each with two replications, followed by two additional replicated tests (Test 3 and 4) for 200 selected ILs from the entire MAGIC population based on the VW resistant performance in the greenhouse. Approximately 10 plants for each line in each replication were grown and screened for VW resistance using a double or triple root inoculation method based on three parameters, i.e., the disease severity rating, percentage of defoliated leaves and percentage of infected plants, with a total of 40,000 plants evaluated. Based on a correlation analysis indicating that the three traits were significantly and positively correlated with one another in each test, the disease severity rating was the best parameter to assess VW resistance due to its relatively low coefficient of variation and its difference in magnitude to differentiate resistant genotypes from susceptible ones. Of the 712 genotypes, 11 showed higher levels of resistance to VW with lower mean disease severity ratings (0.96- 1.33), percent defoliated leaves (7.97-36.31) and percent infected plants (25-100%) across the four tests and these lines are NMIL348, NMIL518, NMIL405, NMIL290, NMIL164, NMIL307, NMIL375, NMIL477, NMIL389, NMIL390, NMIL425, NMIL206, NMIL291, and NMIL398. These VW resistant ILs can be used as parental lines to improve VW resistance in cotton breeding programs.
Genetic Control and Biodiversity of Tolerance to Verticillium Albo-atrum and Verticillium Dahliae in Medicago Truncatula
Author: Azam NegahiPublisher:
ISBN:
Category :
Languages : en
Pages : 0
Book Description
Verticillium wilt, caused by Verticillium albo-atrum (Vaa) and Verticillium dahliae (Vd), is responsible for yield losses in many economically important crops. The capacity of pathogenic fungi to adapt to new hosts is a well-known threat to the durability of resistant crop varieties. Medicago truncatula is a good model for studying resistance and susceptibility to Verticillium wilt in legume plants. Phenotyping a population of inbred lines from a cross between resistant parent line A17 and susceptible parent F83005.5 contributed to the identification of a first QTL controlling resistance to an alfalfa strain of Vaa in M. truncatula. Then, 25 M. truncatula genotypes from a core collection and six Vaa and Vd strains were used to study the potential of non-host Verticillium strains isolated from different plant species to infect this legume plant, and the plant's susceptibility to the pathogens. The experiment was arranged as factorial based on a randomized complete block design with three replications. The wilt symptoms caused by Vaa and Vd were scored on a disease index scale from 0 to 4, during 30 days after inoculation of ten day-old plantlets. Disease severity was quantified by the parameters Maximum Symptom Scores (MSS) and Areas Under the Disease Progress Curves (AUDPC). Highly significant differences were observed among plant genotypes and fungal strains, and their interaction was also significant. The correlation between MSS and AUDPC was 0.86 and highly significant. The most severe symptoms were caused by the alfalfa strain Vaa-V31-2 and the least severe by Vd-JR2, as shown by mean values obtained on the 25 genotypes. M. truncatula genotype TN8.3 was identified as the most susceptible genotype by mean values obtained with the 6 fungal strains, whereas F11013-3, F83005.9 and DZA45.6 were highly resistant to all strains studied. The results were used to choose parents for studying the genetics of resistance in M. truncatula to a nonalfalfa Verticillium strain. So, in the second part of this work, genotype A17 which was susceptible and genotype F83005.5 which was resistant to the potato strain Vaa-LPP0323 and recombinant inbred lines (RILs) from a cross between these genotypes were selected in order to study the genetic control of resistance to this strain of the pathogen. Our experimental design was a randomized complete blocks with 116 RILs and three replications. High genetic variability and transgressive segregation for resistance to Vaa-LPP0323 were observed among RILs. A total of four QTLs controlling resistance to Vaa-LPP0323 were detected for the parameters MSS and AUDPC. The phenotypic variance explained by each QTL (R2) was moderate, ranging from 3 to 21%. A negative sign of additive gene effects showed that favourable alleles for resistance come from the resistant parent.