Author: Peter NeumannPublisher:
ISBN:
Category :
Languages : en
Pages : 300
Book Description
Inter-and Intraspecific Parasitism in Honeybees (Apis Mellifera L.): the Small Hive Beetle (Aethina Tumida Murray) and the Cape Honeybee (A. M. Capensis Esch.)
Author: Peter NeumannEvolution of Intraspecific Social Parasitism in Honeybee Workers (Apis Mellifera Capensis Esch)
Author: Stephan HärtelThe Genetic Control of the Social Parasitism in the Cape Honey Bee, A. M. Capensis ESCH
Author: Denise AumerPublisher:
ISBN:
Category :
Languages : en
Pages :
Book Description
A. m. capensis; social parasitism; thelytoky; reproductive dominance; asexual reproduction
Honeybee Mites and Their Control
Author: International Bee Research AssociationPublisher: Food & Agriculture Org.
ISBN: 9789251025208
Category : Science
Languages : en
Pages : 156
Book Description
Infection in Honey Bees
Author: Giovanni CiliaPublisher: Mdpi AG
ISBN: 9783036529622
Category : Science
Languages : en
Pages : 214
Book Description
Honey -bee pathogens are spread worldwide and are strongly related to the decline of honey bee populations, with which has severe implications for beekeeping, honey production and ecology. Honey bee pathogens are continuously studied by researchers to with the aim to better understand the host--s-parasites relationship of these pathogens and the effects that they have on the bee coloniesy. Honey bee pathogens include bacteria (i.e. i.e., Melissococcus plutonius and Paenibacillus larvae), microsporidia (i.e. i.e., Nosema apis and Nosema ceranae), fungi (i.e. i.e., Ascosphaera apis), protozoa (i.e. i.e., Lotmaria passim, Crithidia bombi and Crithidia mellificae) and viruses (i.e. i.e., ABPV, CBPV, IAPV, KBV, DWV, BQCV and SBV). All of these pathogens are able to infect other bee species; infections would have with important implications in for their life cycles (e.g. e.g., Osmia sp. and Bombus sp.) or cause unknown epidemiological effects for other hymenopterans. BesidesIn addition, old and new invasive pests (such as Varroa destructor, Aethina tumida, Vespa velutina, etc.) necessitate more studies to definite define their role as possible vectors or possible sources of infection for honey bees. For these reasons, knowledge on honey -bee pathogens has become a matter of public interest and is connected with the critical role of honey -bee health. The aim of this Special Issue is to explore honey bee pathogens, considering any aspect in relation to host-pathogen interaction and highlighting the possible interaction and spillover with other bee species and invasive pests, through a series of research articles focusing that focus on different aspects of pathologies.
Tracheal Mite Parasitism of Honey Bees [microform]
![Tracheal Mite Parasitism of Honey Bees [microform] PDF](https://books.google.com/books/content/images/frontcover/mekXnQEACAAJ?fife=w80-h100)
Author: Lynn A. RoycePublisher: Ann Arbor, Mich. : University Microfilms International
ISBN:
Category : Honeybee
Languages : en
Pages : 244
Book Description
The tracheal mite, Acarapis woodi (Rennie), parasitizes the honey bee, Apis mellifera L., an insect used extensively by humans for pollination of domestic crops and for bee products, primarily honey. This parasite was first discovered in 1921 on the Isle of Wight, a small island situated in the English Channel off the southern coast of England. Controversy as to the impact of A. woodi on its honey bee host arose soon after its discovery. This project quantifies the mite-generated mortality of worker honey bees during the flowering period when the bees are active and suggests how mortality of individuals may impact the colony to which these individuals belong. A model of parasitism is derived that demonstrates how swarming may stabilize the populations of host and parasite, how drifting of worker bees between colonies may be directly related to parasite prevalence among the population of colonies and how interruption of swarming may disrupt this system. It appears that honey bee foraging has little impact on mite transmission, but microclimate probably is important. The mites transfer to new hosts mainly during the night. Tracheal mites produce both sexes in sufficient numbers to suggest that parthenogenesis is not invoked to take advantage of quickly expanding resources (i.e. young bees) in the spring. Mite fecundity in the Pacific Northwest peaks in June when bee turnover is greatest. Protein mucopolysaccharide material covers the hemolymph side of infested tracheae and these become darkened with age, suggesting that mite feeding initiates a coagulation-melanization response from the bee. Within a colony uninfested members are also affected by mite infestation of their sisters, as indicated by studies on individual bee physiology.
Nosena Apis
Author: Ingemar FriesPublisher:
ISBN: 9780860982111
Category : Nosema apis
Languages : en
Pages : 19
Book Description
A Molecular Diagnostic Survey of Pathogens and Parasites of Honey Bees, Apis Mellifera L., from Arkansas and Oklahoma
Author: Dylan Alexandra ClearyPublisher:
ISBN:
Category : Honeybee
Languages : en
Pages : 244
Book Description
The health and viability of colonies of the honey bee, Apis mellifera, in the United States have fluctuated dramatically over the past decade. This poses a substantial threat to agricultural production in this country. Currently, no single factor has been identified for this decline. Rather, it has been suggested that the interaction between multiple biotic and abiotic stressors may be responsible. Among these factors are pesticides, habitat loss, climate and weather, parasites and pathogens, and colony management techniques. For this reason, it is important to examine the prevalence of honey bee parasite and pathogen infection at the state level in comparison to national survey data. In the research described herein, molecular diagnostics were performed on worker honey bee samples from Arkansas hobbyist beekeepers and Oklahoma migratory beekeepers to detect the presence of the following A. mellifera pathogens and parasites: protozoans Nosema apis and N. ceranae; bacterial pathogens Spiroplasma apis and S. melliferum; Trypanosomatid parasites Crithidia mellificae and Lotmaria passim and the parasitic phorid fly Apocephalus borealis. A study including both migratory honey bee colonies and hobbyist managed colonies provides a more comprehensive distribution of where these parasite and pathogen species are occurring and potentially why they are occurring. The study determined that N. ceranae (H=11.6%, M=27.6), L. passim (H=11.3%, M=1.1%), and V. destructor (H=45.5%, M=17.2%), occur in both hobbyist and migratory managed colonies. Nosema ceranae was more prevalent in the migratory colonies than the hobbyist colonies. Spiroplasma was also detected in the Oklahoma migratory colony samples (8.05%), but not in the Arkansas hobbyist colonies. Both V. destructor and L. passim were more prevalent in the hobbyist managed colonies. This research resulted in the first detection of Lotmaria passim in Arkansas honey bees, as well as the first documented detection of L. passim and S. melliferum in Oklahoma. Apocephalus borealis, C. mellificae, N. apis, and S. apis were not detected in either the migratory nor the hobbyist colonies. This study compares honey bee management practices at the hobbyist and migratory level to better understand how management influences parasite and pathogen spread and abundance. The use of state-level surveys, when examining parasite and pathogen occurrence, allows for a better understanding of how these pests are spreading, as well as how quickly and by what means.
The Potential Impact of Pathogens on Honey Bee, Apis Mellifera L., Colonies and Possibilities for Their Control
Author: Suresh DesaiPublisher:
ISBN:
Category :
Languages : en
Pages : 0
Book Description
Excessive honey bee colony losses all over the world are believed to be caused by multiple stressors. In this thesis, I characterized and quantified pathogen levels in honey bee colonies, studied their interactions with each other and with their associated parasite vectors, examined factors that influence their combined impacts on honey bees and developed methods to manage honey bee viruses so that colony losses can be minimized. My baseline study of virus prevalence and concentration in healthy and unhealthy (showing visible signs of disease) colonies in Canada showed that seven economically important viruses (DWV, BQCV, IAPV, KBV, SBV, ABPV, and CBPV) were all widely distributed in Canada. Differences in concentration and prevalence of some viruses were found between unhealthy and healthy colonies but these differences may have been due in part to seasonal or regional effects. Studies of the impact of viruses on worker bee populations over winter showed different factors were correlated with bee loss in different environments. Spring concentrations of DWV and mean abundance of Varroa (Varroa destructor) were positively correlated with bee loss and negatively correlated with spring population size in outdoor-wintered colonies. Fall concentration of IAPV was negatively correlated with spring population size of colonies in indoor-wintering environments but not in outdoor-environments. My study showed that it is important to consider location of sampling when associating pathogen loads with bee loss with Nosema and BQCV. Seasonal patterns of parasites and pathogens were characterized for each wintering methods (indoor and outdoor). My results revealed lower ABPV and Nosema ceranae prevalence and lower DWV concentration in genetically diverse than genetically similar colonies. I showed that within colony genetic diversity may be an important evolutionary adaptation to allow honey bees to defend against a wide range of diseases. In laboratory studies, I showed that feeding DWV to larvae in the absence of Varroa causes wing deformity and decreased survival rates of adult bees relative to bees not fed DWV. Finally, I showed that RNA silencing can be used to reduce DWV concentrations in immature and adult bees, reduce wing deformity in emerging adults, and increase their longevity relative to controls.
Author: A. A. M. El-Shemy