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Author: Livio Capocaccia Publisher: CRC-Press ISBN: 9780849389641 Category : Medical Languages : en Pages : 672
Book Description
This is a comprehensive reference covering the advances in hepatic encephalopathy (HE) and metabolic nitrogen exchange. Written by experts in their fields, the text discusses various aspects of hepatic nitrogen metabolism, protein metabolism, renal ammonia metabolism, neurobiology and neuropharmacology, and ammonia in the pathogenesis of HE. Benzodiazepine receptor ligands in the pathogenesis of HE are featured, and amino acids, amines, and zinc in the pathogeneis of HE are investigated. Studies done with various animal models are included. New techniques, MR spectroscopy and imaging, are highlighted. Steps for the diagnosis and assessment of HE are provided, and the effects of therapy are examined. Useful information is given regarding the metabolic aspects of liver cirrhosis and liver cell physiology.
Author: Robert S. Brown Jr Publisher: Elsevier Health Sciences ISBN: 0323376037 Category : Medical Languages : en Pages : 161
Book Description
Dr. Brown provides a timely upate on the clinical diagnosis and management of Hepatic Encephalopathy. His issue addresses current hot topics including Role of NH4 in Pathogenesis of Hepatic Encephalopathy; How to diagnose and manage HE in fulminant hepatic failure; New methods of Brain Imaging and testing (eg PET) and Hepatic Encephalopathy; Management New agents for HE (focus on NH4 lowering drugs used in UCD); Legal responsibility of MD when he diagnoses HE; Diets in Encephalopathy; and Neurological manifestations of HE.
Author: Vicente Felipo Publisher: Springer Science & Business Media ISBN: 1461519896 Category : Medical Languages : en Pages : 215
Book Description
This volume contains the papers presented at the International Symposium on "Cirrhosis, Hyperammonemia and Hepatic Encephalopathy", held in Valencia, Spain, January 24th-27th, 1994. Liver cirrhosis and other hepatic dysfunctions such as fulminant hepatic failure and congenital defects of urea cycle enzymes can lead to hepatic encephalopathy, coma and death. Hepatic encephalopathy is one of the main causes of death in western countries. The ability to detoxify ammonia by its incorporation into urea is diminished by impaired liver function, resulting in increased ammonia levels in blood and brain. Hyperammonemia is considered one of the main factors in the mediation of hepatic encephalopathy and the classical clinical treatments are directed towards reducing blood ammonia levels. However, the molecular bases of the pathogenesis of hepatic encephalopathy and the role of hyperammonemia in this process remain unclear and several hypotheses have been proposed. To clarify the mechanisms involved in hepatic encephalopathy and hyperammonemia suitable animal models are necessary. The animal models available and the ideal features of an animal model are presented in the initial part of the book.
Author: E. Anthony Jones Publisher: Springer Science & Business Media ISBN: 9401001596 Category : Medical Languages : en Pages : 454
Book Description
Encephalopathy and Nitrogen Metabolism in Liver Failure contains research reports and state-of-the-art reviews that were presented at the 11th International Symposium on Hepatic Encephalopathy and Nitrogen Metabolism in relation to liver disease, that was held in Amsterdam, The Netherlands from 30 May to 1 June, 2002. Themes covered include the metabolism of ammonia and amino acids in relation to liver disease, the assessment of hepatic encephalopathy (HE), minimal HE, animal models of HE, neuropsychiatric dysfunction in patients with chronic liver disease, the pathogenesis of HE, the astrocyte in liver disease, acute liver failure and cerebral edema, treatment of HE and artificial liver support systems. This volume provides an indispensable source of important new research findings and ideas for clinicians and biomedical scientists working in the field of HE and nitrogen metabolism in relation to liver disease.